What is the doc for torsades de pointes?
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What is the doc for torsades de pointes?
Intravenous magnesium sulfate, a relatively new mode of therapy for torsade de pointes, was proven to be extremely effective and is now regarded as the treatment of choice for this arrhythmia.
What does torsades turn into?
Most cases of torsades de pointes resolve on their own without treatment. However, it can develop into ventricular fibrillation, which can lead to cardiac arrest and may even be fatal.
How do you fix Torsades de Pointes?
Pulseless torsades should be defibrillated. Intravenous magnesium is the first-line pharmacologic therapy in Torsades de Pointes. Magnesium has been shown to stabilize the cardiac membrane, though the exact mechanism is unknown. The recommended initial dose of magnesium is a slow 2 g IV push.
How do you identify torsades de pointes on an ECG?
Torsades de pointes is a specific form of polymorphic ventricular tachycardia in patients with a long QT interval. It is characterized by rapid, irregular QRS complexes, which appear to be twisting around the electrocardiogram (ECG) baseline.
Why is magnesium used in torsades?
Answer. Magnesium is the drug of choice for suppressing early afterdepolarizations (EADs) and terminating the arrhythmia. Magnesium achieves this by decreasing the influx of calcium, thus lowering the amplitude of EADs.
What type of rhythm is torsades de pointes?
Torsades de Pointes is a type of very fast heart rhythm (tachycardia) that starts in your heart’s lower chambers (ventricles). Unlike a normal pulse rate of 60 to 100 beats a minute, a fast heartbeat in your ventricles (ventricular tachycardia) is more than 100 beats a minute.
Do you give amiodarone for torsades?
Lidocaine is the preferred antiarrhythmic drug for torsade, although there isn’t a ton of evidence supporting its use. Do not use amiodarone, procainamide, beta-blockers, or most other antiarrhythmics.
Why does long QT lead to torsades?
In the long QT syndromes (LQTS), malfunction of ion channels impairs ventricular repolarisation and triggers a characteristic ventricular tachyarrhythmia: torsade de pointes. Symptoms in the LQTS (syncope or cardiac arrest) are caused by this arrhythmia.
How does mag sulfate help torsades?
Magnesium sulphate suppresses torsade by decreasing the influx of calcium ions, which in turn results in decreased amplitude of EADs. The initial dose is 2 g (20 ml of 10% solution), given intravenously over one to two minutes.
Can potassium cause QT prolongation?
Potassium levels below 3,0 mmol/l cause significant Q-T interval prolongation with subsequent risk of torsade des pointes, ventricular fibrillation and sudden cardiac death. Potassium levels above 6,0 mmol/l cause peaked T waves, wider QRS komplexes and may result in bradycardia, asystole and sudden death.
What is the difference between Torsades and polymorphic VT?
Polymorphic VT is defined as an unstable rhythm with a continuously varying QRS complex morphology in any recorded ECG lead. Polymorphic VT that occurs in the setting of QT prolongation is considered as a distinct arrhythmia, known as torsades de pointes.
Does a pacemaker prevent torsades?
Pacemaker reprogramming to 100 beats/min prevented further triggering of torsades de pointes. Other factors that may trigger torsades de pointes (such as hypokalaemia and antiarrhythmic drugs) were excluded.
Why is magnesium used in torsades de pointes?
Why is magnesium used to treat torsades?
