What is Postobstructive diuresis?

What is Postobstructive diuresis?

By definition, post-obstructive diuresis is the condition of prolonged urine production of at least 200 cc for at least two consecutive hours immediately following the relief of urinary retention or similar obstructive uropathy. It may also be defined as more than 3,000 cc over 24 hours.[6]

Can Hypernatremia cause polyuria?

Polyuria and polydipsia. More mild cases of hypernatremia (plasma Na+ concentrations 144 – 149 meq/L) may present in patients who are awake and alert but complain of polyuria defined as > 3L urine per day. Diagnosis can be made using the water restriction test.

How long does Postobstructive diuresis last?

Physiologic POD is self-limiting and generally lasts 24 hours. Pathologic POD generally lasts longer than 48 hours and can be exacerbated with excessive intravenous fluid replacement. The treatment of urinary retention begins with immediate catheter placement to decompress the bladder.

How is post obstructive diuresis treated?

Treatment of postobstructive diuresis consists of judicious fluid replacement with 0.45% saline (at a rate slightly less than urine output) and replacement of electrolytes. Urinary tract infections may occur due to urinary stasis. Additionally, instrumentation may also introduce contamination.

What is Postobstructive pneumonia?

Postobstructive pneumonia is defined as an infection of lung parenchyma secondary to bronchial obstruction (4). It is often associated with lung malignancy (see Figure 1).

What is Diurese?

Definition. Diuresis is a condition in which the kidneys filter too much bodily fluid. That increases your urine production and the frequency with which you need to use the bathroom. Most adults will urinate about four to six times a day, with average output between 3 cups and 3 quarts of urine.

How hypercalcemia causes polyuria?

Up to 20% of patients with hypercalcemia develop polyuria. The postulated mechanism is downregulation of aquaporin-2 water channels, and calcium deposition in the medulla with secondary tubulointerstitial injury, leading to impaired generation of the interstitial osmotic gradient.

Does polyuria cause hyponatremia?

Volume contraction and elevated angiotensin II levels stimulate the release of antidiuretic hormone, which results in thirst and polydipsia. The antidiuretic hormone activity, along with excess free water intake from polydipsia relative to urine sodium wasting, results in hyponatremia.

Is urinary blockage an emergency?

People with acute urinary retention cannot urinate at all, even though they have a full bladder. Acute urinary retention, a potentially life-threatening medical condition, requires immediate emergency treatment.

What is the ICD 10 code for Postobstructive pneumonia?

Pneumonia due to other specified infectious organisms The 2022 edition of ICD-10-CM J16. 8 became effective on October 1, 2021.

What is Postobstructive pulmonary edema?

Postobstructive pulmonary edema is a well-recognized complication of upper airway obstruction. The mechanisms of edema formation are unclear and may be due to increased hydrostatic forces generated by high negative inspiratory pressure or by increased permeability of the alveolar capillary membrane.

What is the difference between diuresis and natriuresis?

Natriuresis: The excretion of an excessively large amount of sodium in the urine. Natriuresis is similar to diuresis (the excretion of an unusually large quantity of urine), except that in natriuresis the urine is exceptionally salty.

When do you Diurese a patient?

So suffice to say that it’s time to diurese when the patient’s current fluid status becomes a hindrance more than a help. Note that this has little to do with their absolute balance, and some patients may actually be healthiest while either more positive or more negative than they were admitted.

Does hypocalcemia cause polyuria?

Therefore, potassium deficiency may lead to concentrating defects via this pathway, leading to polyuria. There is also some evidence that hypokalaemia leads to impaired responsiveness to ADH. The exact mechanism is unclear, but obviously decreased responsiveness to ADH may lead to large volumes of dilute urine.

How does hypercalcemia affect urine concentration?

Hypercalcemia increased urinary excretion of iCa, iMg, sodium, phosphate, potassium, and chloride; increased urine output; and decreased urine osmolality and specific gravity.

What is the differential diagnosis of polyuria?

It can result from any of three basic defects: (a) inadequate urinary concentration caused by a deficiency in the secretion or action of the antidiuretic hormone vasopressin (neurogenic or nephrogenic DI), or excessive intake of water caused by a defect in (b) thirst or (c) psychological function (dipsogenic or …