What is the difference between narcolepsy type 1 and 2?

What is the difference between narcolepsy type 1 and 2?

Type 1 narcolepsy (previously termed narcolepsy with cataplexy). This diagnosis is based on the individual either having low levels of a brain hormone (hypocretin) or reporting cataplexy and having excessive daytime sleepiness on a special nap test. Type 2 narcolepsy (previously termed narcolepsy without cataplexy).

How is narcolepsy with cataplexy different from REM sleep?

Some of the characteristics of narcolepsy — such as cataplexy, sleep paralysis and hallucinations — are similar to changes that occur in REM sleep, but instead, they occur during wakefulness or drowsiness.

Which type of narcolepsy is more common?

There are two types of narcolepsy: Type 1 is the most common. It includes a symptom called cataplexy, or sudden loss of muscle tone.

Which is worse type 1 or 2 narcolepsy?

In general, the symptoms of type 2 narcolepsy tend to be less severe than those of type 1 narcolepsy. In addition to EDS, type 2 narcolepsy may cause: sleep paralysis. hallucinations.

Is narcolepsy type 2 an autoimmune disease?

Studies in recent years suggest that narcolepsy may be an autoimmune disorder, primarily triggered by a genetic mutation. Though narcolepsy is a chronic condition that currently has no cure, researchers are working to learn more about its causes and how to safely and effectively treat it.

Do I have type 2 narcolepsy?

Type 2 narcolepsy (previously called narcolepsy without cataplexy). Persons with type 2 narcolepsy have excessive daytime sleepiness but do not have cataplexy and have normal levels of hypocretin.

Can narcolepsy be psychosomatic?

Narcolepsy as a Type of Response to Emotional Conflicts : Psychosomatic Medicine.

Can anxiety mimic narcolepsy?

Anxiety. Over half of people who have narcolepsy also have an anxiety disorder like panic attacks or social anxiety. You could also become anxious as a response to the hallucinations (seeing or hearing things that aren’t there) or cataplexy (sudden muscle weakness) that narcolepsy can cause.

What is the difference between EDS and narcolepsy?

EDS means that you get overcome by an irresistible need to sleep during the day, and you can feel tired all the time. People with narcolepsy can fall asleep randomly and at unexpected times, even while eating or talking with someone.

What is pseudo cataplexy?

Pseudo-cataplexy events are characterized by negative emotional triggers such as crying, generalized weakness, abrupt onset, preserved ability to communicate, and preserved deep tendon reflexes. Pseudo-cataplexy has been associated with mood disorders.

What are the symptoms of Kleine Levin syndrome?

Symptoms occur as “episodes,” typically lasting a few days to a few weeks. Episode onset is often abrupt, and may be associated with flu-like symptoms. Excessive food intake, irritability, childishness, disorientation, hallucinations, and an abnormally uninhibited sex drive may be observed during episodes.

How is secondary narcolepsy treated?

There is no known cure for narcolepsy, so treatment is primarily targeted at managing symptoms (e.g., daytime sleepiness, cataplexy). Non-pharmacologic treatments include patient education, sleep hygiene, daytime naps, and changes in lifestyle as necessary.

What can be misdiagnosed as narcolepsy?

Narcolepsy is often misdiagnosed as other conditions that can have similar symptoms, including:

• Depression.
• Anxiety.
• Other psychologic/psychiatric disorders.
• Insomnia.
• Obstructive sleep apnea.

What is Pseudosleep?

Preictal pseudosleep was defined as a state that resembled normal sleep by behavioral criteria alone (i.e. patient motionless and eyes closed), while EEG showed evidence of wakefulness (alpha rhythm, active EMG, and rapid eye movement). This state had to be sustained for at least 1 minute before clinical onset.

What is Kevin syndrome?

Kleine-Levin syndrome is a rare disorder characterized by the need for excessive amounts of sleep (hypersomnolence), (i.e., up to 20 hours a day); excessive food intake (compulsive hyperphagia); and behavioral changes such as an abnormally uninhibited sexual drive.